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Preclinical work has reported a distinct selectivity profile, with growth hormone stimulation observed at concentrations that did not trigger ACTH or cortisol secretion, making it a useful tool for isolating growth hormone-specific signalling without activation of the hypothalamic-pituitary-adrenal axis

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While these medications provide a powerful exogenous boost to satiety hormones, they often function as a chemical Band-Aid masking symptoms of a broken appetite-regulation system without fixing the underlying biology